Cordycepin Powder

Cordycepin, also known as cordycepin, cordycepin, and cordycepin, has a chemical formula of C10H13N5O3 and is the first nucleoside antibiotic isolated from fungi.

Cordyceps sinensis polysaccharide is a confirmed source, polysaccharides, and a large host of other newly discovered nutritional compounds, Cordyceps sinensis polysaccharide based tonic that boosted levels of energy, vitality, endurance and stamina in the body.

1. Lung and kidney protection
In the study of western medicine, Ma Shumei and others found that cordycepin can effectively reduce pulmonary fibrosis, improve lung tissue structure and pathological changes of fibrosis. Zhang Min and others found that a certain dose of cordycepin inhibited the chronic airway inflammation of asthma by adopting a rat model of bronchial asthma, and at the same time combined with glucocorticoids could effectively inhibit the contraction of airway smooth muscle, reduce the occurrence of asthma, and reduce the incidence of asthma. Airway remodeling has a certain improvement effect. Cordycepin inhibits the production of cytokines in COPD (chronic obstructive pulmonary disease), reduces inflammation around lung organs and blood vessels, and relieves pulmonary pathological changes such as epithelial hyperplasia.
2. Anti-three high effects
Three highs are the general term for hyperlipidemia, high blood pressure and high blood sugar. According to research, the aggregation of these three clinical diseases is not accidental, and the three affect each other in pairs. Studies have shown that cordycepin has a good effect on lowering blood lipids, and it also affects fat production while lowering blood lipids. In 2008, Zhao Haiyan and others found that cordycepin can have a certain protective effect on pancreatic cells under high glucose. In the treatment of early atherosclerosis, cordycepin inhibited the inflammatory response and protected the integrity of the vascular endothelium.
3. Anti-tumor effect
At present, cancer is one of the high-incidence diseases of human beings, which has caused people to "talk about cancer". In recent years, research on the anticancer activity and mechanism of action of cordycepin has become a hot topic. Park et al. determined the antitumor activity of cordycepin by using the method of measuring protein level-immunoblotting, and found that the number of apoptotic cells increased significantly in the comparison between the treatment group and the control group. At the same time of cell apoptosis, C6 glioma The migration and invasion abilities of cells were also reduced, and the research results indicated that cordycepin may inhibit the proliferation of cancer cells by regulating p85/AKT or GSK3β-r. Kim et al. found that the fermented cordycepin mycelia liquid had a stronger inhibitory effect on melanoma. It can inhibit the proliferation of melanoma by promoting the expression of Bax apoptosis protein in melanoma cells.

4. Nerve protection and regulation
Cordycepin has the function of neuroprotection and regulation, and can effectively protect animal neurons. Jin et al. found that cordycepin can reduce the increase of ROS and Ca2+ levels caused by glutamate, and its anti-apoptotic effect is partly dependent on the activation of adenosine A1 receptors. The results indicate that cordycepin may be a potential therapeutic agent for neuronal diseases . Chen et al. found that cordycepin has an important neuroprotective effect in hypoxic or ischemic injury. In the process of studying the electrophysiological mechanism of cordycepin to protect ischemia and hypoxic injury, it was found that cordycepin (80 μmol/L) can significantly delay hypoxia-induced membrane depolarization, prolong the ability of spontaneous action potential (AP) firing, delay rapid depolarization occurs. In addition, cordycepin also delayed the decline of the induced AP amplitude caused by hypoxia, and studies have proved that cordycepin has an important neuroprotective effect against hypoxic injury by improving the electrophysiological function of neurons. At present, neuroinflammation has been considered as the main cause of cerebral hemorrhage and brain injury. Cheng et al. induced cerebral hemorrhage in male mice by stereotaxic injection of autologous blood, and then intraperitoneally administered cordycepin to mice after successful induction of cerebral hemorrhage, and found that cordycepin could significantly relieve neurological deficits after cerebral hemorrhage. Functional deficits, cerebral edema, and tissue damage around the hematoma. In addition, cordycepin improved neuronal death in the hematoma area, suggesting that cordycepin played a neuroprotective role in the ICH model. Meng Xuelian found that cordycepin can inhibit the production of NO in microglia, protect PC12 neurons, and protect PC12 cells damaged by H2O2 by increasing SOD activity.

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